Congratulations to Andreas Mathisen, our PhD student, for obtaining one year mobility fellowship from Faculty of Medicine, University of Bergen, to study and work at University of Geneva, Switzerland! We are proud of you!
We are very happy to be partners on a new research project financed under the EEA and Norway Grants scheme promoting research collaboration between Romania and Norway. Our collaborator, Dr. Ana Vacaru from the Institute for Cellular Biology and Pathology…
Congratulations to Andreas Mathisen, who has received a 4-years PhD fellowship from the Faculty of Medicine, UiB. #PhDlife Andreas will start his project early 2020 working on complex genetic setups in order to dynamically characterize the surviving β-cell feedback to…
The Nordic Developmental Biology organisations are announcing travel grants of 6000 SEK each, to help its members at the postdoctoral and graduate student level to attend courses, meetings or workshops during the year 2019 that promote their research and education…
The Norwegian Research Council announced on 7th of December the new list of awardees. We got new funding from FRIPRO for another independent Young Research Talent project: “Characterizing and modulating the insulin-producing beta-cell fate in monogenic diabetes by using novel…
The second project of the lab is funded by the Research Council of Norway, as a Young Talent Research Project. This 4-year project is aiming at characterizing the cellular and molecular basis of the gradual failure of insulin-producing β-cells in…
Diabetes mellitus is a group of metabolic diseases defined by high blood sugar values caused by the inability of the body to produce and/or use insulin. All forms of diabetes are ultimately characterized by a decrease in the number of functional insulin-producing cells (β-cells), hence a cure for insulin-dependent diabetes types will require their regeneration or replacement. Generally, the most efficient regenerative strategies are the ones involving cell self-renewal capacity. Nevertheless, in mammals, the β-cell proliferative capacity is very low after birth and decreases even further with age. The overall aim of this proposal is to elucidate and reverse the molecular age-switch controlling the gradual impairment of β-cell self-renewal potential by using two murine models of monogenic diabetes.