Ulrik Larsen


SCIENTIFIC CAREER


Since 2023

PhD Candidate at Center for Diabetes Research, Department of Clinical Science, Faculty of Medicine, University of Bergen, Norway

2021 – 2023 Master in Biomedical Sciences, Faculty of Medicine, University of Bergen, Norway

2018 – 2021 Bachelor in Biochemistry, Faculty of Science and Technology, University of Stavanger, Norway


PUBLICATIONS while in the lab


  1. Hossain MK, Unger L, Larsen U, Altankhuyag A, Legøy TA, Paulo JA, Vethe H, Ghila L. Mapping the initial effects of carcinogen-induced oncogenic transformation in the mouse bladder. Exp Cell Res. 2025 Feb 21:114452. doi: 10.1016/j.yexcr.2025.114452.
  2. Mathisen AF, Larsen U, Kavli N, Unger L, Vacaru AM, Daian LM, Vacaru A-M, Herrera PL, Ghila L, Chera S. Moderate beta-cell ablation triggers synergic compensatory mechanisms even in the absence of overt metabolic disruption. Commun Biol 7, 833 (2024). https://doi.org/10.1038/s42003-024-06527-5
  3. Mathisen AF, Legøy TA, Larsen U, Unger L, Abadpour S, Paulo JA, Scholz H, Ghila L, Chera S. The age-dependent regulation of pancreatic islet landscape is fueled by a HNF1a-immune signaling loop. Mech Ageing Dev. 2024 May 31:111951. doi: 10.1016/j.mad.2024.111951

ORAL Presentations


  1. Ulrik Larsen, Andreas Froslev Mathisen, Natalie Kavli, Luiza Ghila, Simona Chera. Partial insulin-producing β-cell ablation induces compensatory proliferation in mouse pancreatic islets. MRCRM 2nd International Meeting on Regenerative Medicine 15-16 April 2024 Bergen, Norway

POSTER presentations


  1. Ulrik Larsen, Andreas Froslev Mathisen, Natalie Kavli, Luiza Ghila, Simona Chera. Investigating stress-induced islet cell proliferation following partial insulin-secreting β-cell ablation in mice. The Nordic Developmental Biology Societies & Michael Sars Symposium Joint Meeting, 28-31 May 2024, Bergen, Norway

EMBO COURSE


  1. EMBO Practical Course Quantitative proteomics: strategies and tools to probe biology, 9-14 March 2025, EMBL Heidelberg, Germany 

Journal Club Posts


  1. The Colon is an Extra-Pancreatic Source of Glucagon: Demonstration in a Mouse Model of Pancreatectomy