PhD candidate at Department of Clinical Science, Faculty of Medicine, University of Bergen, Norway
2023 – 2024 Researcher at Department of Clinical Science, Faculty of Medicine, University of Bergen, Norway
2021 – 2023 Master in Biomedical Sciences, Faculty of Medicine, University of Bergen, Norway
2019 – 2021 Undergraduate Teaching Assistant, Department of Pharmacy, Brac University, Bangladesh
2015 – 2019 Bachelor in Pharmacy, Department of Pharmacy, Brac University, Bangladesh
PUBLICATIONS while in the lab
Unger L, Larsen U, Legøy TA, Hossain MK, Gudmestad JH, Friestad A, Sharmine S, Njølstad PR, Ræder H, Herrera PL, Ghila L, Chera S. Defective HNF1A hinders GLI3 processing favouring duodenal versus pancreatic fate thus leading to intestinal elongation in vivo. Genes & Development 2026 Mar 26. doi: 10.1101/gad.353153.125.
Unger L, Larsen U, Sharmine S, Hossain MK, Legøy TA, Vaudel M, Ghila L, Chera S. Efficient cytoplasmic cell quantification using a semi-automated FIJI-based tool. Sci Rep. 2025 Jul 28;15(1):27509. doi: 10.1038/s41598-025-12144-x.
Hossain MK, Unger L, Larsen U, Altankhuyag A, Legøy TA, Paulo JA, Vethe H, Ghila L. Mapping the initial effects of carcinogen-induced oncogenic transformation in the mouse bladder. Exp Cell Res. 2025 Feb 21:114452. doi: 10.1016/j.yexcr.2025.114452.
Molecular rewiring leading to bladder cancer tumorigenesis. CCBIO’s 14th Annual Symposium, 12-13 May 2026, Solstrand, Norway
Mapping the effects of carcinogen-induced oncogenic transformation in the mouse bladder. CCBIO’s 13th Annual Symposium, 13-14 May 2025, Solstrand, Norway
Short-term BBN treatment alters the proteomic profile of mouse bladder. CCBIO’s 12th Annual Symposium, 14-15 May 2024, Solstrand, Norway
POSTER presentations
Md Kaykobad Hossain, Lucas Unger, Ulrik Larsen, Altanchimeg Altankhuyag, Thomas Aga Legøy, Joao A Paulo, Heidrun Vethe, Luiza Ghila. Molecular rewiring leading to bladder cancer tumorigenesis. CCBIO’s 14th Annual Symposium, 12-13 May 2026, Solstrand, Norway
Md Kaykobad Hossain, Lucas Unger, Ulrik Larsen, Altanchimeg Altankhuyag, Thomas Aga Legøy , Joao A Paulo, Heidrun Vethe, Luiza Ghila. Mapping the effects of carcinogen-induced oncogenic transformation in the mouse bladder. 61st Contact Meeting of the Norwegian Bioscience Society 21-24 January 2026 Gol, Norway
Md Kaykobad Hossain, Lucas Unger, Luiza Ghila. Understanding mouse bladder tumorigenesis at single-cell resolution. c-myc Single Cell Satellite Seminar, 12th May 2025, Solstrand, Norway
Md Kaykobad Hossain, Lucas Unger, Ulrik Larsen, Altanchimeg Altankhuyag, Thomas Aga Legøy , Joao A Paulo, Heidrun Vethe, Luiza Ghila. Mapping the effects of carcinogen-induced oncogenic transformation in the mouse bladder. CCBIO’s 13th Annual Symposium, 13-14 May 2025, Solstrand, Norway
Md Kaykobad Hossain, Altanchimeg Altankhuyag, Thomas A. Legøy, Simona Chera, Luiza Ghila. Dynamic proteome profiling detects progressive alterations induced by BBN in the mouse bladder. The Nordic Developmental Biology Societies & Michael Sars Symposium Joint Meeting, 28-31 May 2024, Bergen, Norway
Md Kaykobad Hossain, Altanchimeg Altankhuyag, Thomas A. Legøy, Simona Chera, Luiza Ghila. Short-term BBN treatment alters the proteomic profile of mouse bladder. CCBIO’s 12th Annual Symposium, 14-15 May 2024, Solstrand, Norway
Md Kaykobad Hossain, Altanchimeg Altankhuyag, Thomas A. Legøy, Simona Chera, Luiza Ghila. Mapping predictive bladder cancer signatures in a mouse model. CCBIO’s 11th Annual Symposium, 8-10 May 2023, Solstrand, Norway
FUNDING
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Period
Grant Description
2025-2026
Meltzer prosjektstipend: Deciphering the role of WNT4 in bladder cancer using patient-derived organoids We plan to investigate the role of Wnt4 in the generation of PDOs, including an assessment of the tumorigenic ability of laboratory grown PDOs by injecting them into mice and by observing tumor formation.
2024-2028
PhD Project: Investigating cell transitions and predictive cancer signature in bladder urothelium We plan to use complex genetic setups, patient derived organoids and multi-omics to dynamically characterize the switch from epithelial regeneration to tumorigenesis using bladder urothelium as a model.
